Psoriatic arthritis

Watad A et al. Curr Rheumatol Rep 2018; 20: 41. doi: 10.1007/s11926-018-0751-3

Feld J et al. Nat Rev Rheumatol 2018; 14: 363–371. doi: 10.1038/s41584-018-0006-8

Veale DJ et al. Rheumatology (Oxford) 2018; Advance online publication. doi: 10.1093/rheumatology/key070

Cafaro G, McInnes IB. Clin Rheumatol 2018; Advance online publication. doi: 10.1007/s10067-018-4012-7

Nat Rev Rheumatol 2018; 14: 170–180. doi: 10.1038/nrrheum.2018.17

In this review, the authors discuss three interconnected mechanisms that might be involved in the predilection of specific joints for developing specific forms of arthritis.

Nat Rev Rheumatol 2017;13:433–442. doi:10.1038/nrrheum.2017.96

Lymphocytes and dendritic cells play an important role in the pathogenesis of psoriatic arthritis. This chapter explores their role in the early and late phase of the disease.

de Vlam K. In: Psoriatic Arthritis and Psoriasis. Edited by Adebajo A et al. Springer International Publishing Switzerland, 2016. doi:10.1007/978-3-319-19530-8_7

A review of advances in the understanding of bone homeostasis and abnormalities in the involved biological pathways in psoriatic arthritis, including the discovery of the interleukin-23/T helper 17 pathway.

Haroon N, Ritchlin C. In: Psoriatic Arthritis and Psoriasis. Edited by Adebajo A et al. Springer International Publishing Switzerland, 2016. doi:10.1007/978-3-319-19530-8_13

Coates et al. discuss the etiology of psoriatic arthritis, including genetic and environmental factors. Various inflammatory pathways are also briefly discussed in this chapter.

In: Handbook of Psoriatic Arthritis and Psoriasis. Edited by Warren R, Menter A. Springer International Publishing Switzerland, 2016. doi:10.1007/978-3-319-18227-8_2

In this review, Fitzgerald et al discuss psoriatic arthritis pathogenesis in light of studies of human leukocyte antigen genotypes and a study on the relationship of genotype to phenotype.

FitzGerald O et al. Arthritis Res Ther 2015;17:115. doi:10.1186/s13075-015-0640-3

A regulatory role of T helper 17 (Th17) cells has been proposed in psoriatic arthritis (PsA). This review discusses the contributing role of the interleukin (IL)-23/IL-17 axis in the pathogenesis of PsA.

Raychaudhuri SK, Saxena A, & Raychaudhuri SP. Clin Rheumatol 2015;34:1019–1023. doi:10.1007/s10067-015-2961-7